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  1. nope i tend to fully agree, now if we take our friendly GPs they used to go from home to home treating elderly and young alike, Drs never became ill seeing to all these patients, hows that work ? think as ye be, your thoughts are powerful for fact
  2. hi Tom think they can just root out the book section on this site, if not I can provide pdfs.
  3. yes bro my teacher taught pretty much that 70% alkaline 30% acid is a perfect human balance.
  4. foreword copied evidence of world wide scam. The book Virus Mania by Torsten Engelbrecht and Claus Kohnlein Foreword I The Content of This Book Has To Be Read, Quickly and Worldwide The book Virus Mania by Torsten Engelbrecht and Claus Kohnlein presents a tragic message that will, hopefully, contribute to the re-insertion of ethical values in the conduct of virus research, public health policies, media communications, and activities of the pharmaceutical companies. Obviously, elementary ethical rules have been, to a very dangerous extent, neglected in many of these fields for an alarming number of years. When American journalist Celia Farber courageously published, in Harper's Magazine (March 2006) the article "Out of control-AIDS and the corruption of medical science," some readers probably attempted to reassure themselves that this "corruption" was an isolated case. This is very far from the truth as documented so well in this book by Engelbrecht and Kohnlein. It is only the tip of the iceberg. Corruption of research is a widespread phenomenon currently found in many major, supposedly contagious health problems, ranging from AIDS to Hepatitis C, Bovine spongiform encephalopathy (BSE or "mad cow disease"), SARS, Avian flu and current vaccination practices (human papillomavirus or HPV vaccination). In research on all of these six distinct public health concerns scientific research on viruses (or prions in the case of BSE) slipped onto the wrong track following basically the same systematic pathway. This pathway always includes several key steps: inventing the risk of a disastrous epidemic, incriminating an elusive pathogen, ignoring alternative toxic causes, manipulating epidemiology with non-verifiable numbers to maximize the false perception of an imminent catastrophe, and promising salvation with vaccines. This guarantees large financial returns. But how is it possible to achieve all of this? Simply by relying on the most powerful activator of human decision making process, i.e. FEAR! We are not witnessing viral epidemics; we are witnessing epidemics of fear. And both the media and the pharmaceutical industry carry most of the responsibility for amplifying fears, fears that happen, incidentally, to always ignite fantastically profitable business. Research hypotheses covering these areas of virus research are practically never scientifically verified with appropriate controls. Instead, they are established by "consensus." This is then rapidly reshaped into a dogma, efficiently 11 Foreword I perpetuated in a quasi-religious manner by the media, including ensuring that research funding is restricted to projects supporting the dogma, excluding research into alternative hypotheses. An important tool to keep dissenting voices out of the debate is censorship at various levels ranging from the popular media to scientific publications. We haven't learnt well from past experiences. There are still many unanswered questions on the causes of the 1918 Spanish flu epidemic, and on the role of viruses in post-WWII polio (DDT neurotoxicity?). These modern epidemics should have opened our minds to more critical analyses. Pasteur and Koch had constructed an understanding of infection applicable to several bacterial diseases. But this was before the first viruses were actually discovered. Transposing the principles of bacterial infections to viruses was, of course, very tempting but should not have been done without giving parallel attention to the innumerable risk factors in our toxic environment; to the toxicity of many drugs, and to some nutritional deficiencies. Cancer research had similar problems. The hypothesis that cancer might be caused by viruses was formulated in 1903, more than one century ago. Even today it has never been convincingly demonstrated. Most of the experimental laboratory studies by virus-hunters have been based on the use of inbred mice, inbred implying a totally unnatural genetic background. Were these mice appropriate models for the study of human cancer? (we are far from being inbred!) True, these mice made possible the isolation and purification of "RNA tumor viruses," later renamed "retroviruses" and well characterized by electron microscopy. But are these viral particles simply associated with the murine tumors, or are they truly the culprit of malignant transformation? Are these particles real exogenous infective particles, or endogenous defective viruses hidden in our chromosomes? The question is still debatable. What is certain is that viral particles similar to those readily recognized in cancerous and leukemic mice have never been seen nor isolated in human cancers. Of mice and men ... However, by the time this became clear, in the late 1960s, viral oncology had achieved a dogmatic, quasi-religious status. If viral particles cannot be seen by electron microscopy in human cancers, the problem was with electron microscopy, not with the dogma of viral oncology! This was the time molecular biology was taking a totally dominant posture in viral research. "Molecular markers" for retroviruses were therefore invented (reverse transcriptase for example) and substituted most conveniently for the absent viral particles, hopefully salvaging the central dogma of viral oncology. This permitted the viral hypothesis to survive for another ten years, until the late 1970s, with the help of increasingly generous support from funding agencies and from pharmaceutical companies. However by 12 Foreword I 1980 the failure of this line of research was becoming embarrassingly evident, and the closing of some viral oncology laboratories would have been inevitable, except that ... Except what? Virus cancer research would have crashed to a halt except that, in 1981, five cases of severe immune deficiencies were described by a Los Angeles physician, all among homosexual men who were also all sniffing amyl nitrite, were all abusing other drugs, abusing antibiotics, and probably suffering from malnutrition and STDs (sexually transmitted diseases). It would have been logical to hypothesize that these severe cases of immune deficiency had multiple toxic origins. This would have amounted to incrimination of these patients' life-style. Unfortunately, such discrimination was, politically, totally unacceptable. Therefore, another hypothesis had to be found-these patients were suffering from a contagious disease caused by a new ... retrovirus! Scientific data in support of this hypothesis was and, amazingly enough, still is totally missing. That did not matter, and instantaneous and passionate interest of cancer virus researchers and institutions erupted immediately. This was salvation for the viral laboratories where AIDS now became, almost overnight, the main focus of research. It generated huge financial support from Big Pharma, more budget for the CDC and NIH, and nobody had to worry about the life style of the patients who became at once the innocent victims of this horrible virus, soon labeled as HIV. Twenty-five years later, the HIV 1 AIDS hypothesis has totally failed to achieve three major goals in spite of the huge research funding exclusively directed to projects based on it. No AIDS cure has ever been found; no verifiable epidemiological predictions have ever been made; and no HIV vaccine has ever been successfully prepared. Instead, highly toxic (but not curative) drugs have been most irresponsibly used, with frequent, lethal side effects. Yet not a single HIV particle has ever been observed by electron microscopy in the blood of patients supposedly having a high viral load ! So what? All the most important newspapers and magazine have displayed attractive computerized, colorful images of HIV that all originate from laboratory cell cultures, but never from even a single AIDS patient. Despite this stunning omission the HIV I AIDS dogma is still solidly entrenched. Tens of thousands of researchers, and hundreds of major pharmaceutical companies continue to make huge profits based on the HIV hypothesis. And not one single AIDS patient has ever been cured ... Yes, HIV I AIDS is emblematic of the corruption of virus research that is remarkably and tragically documented in this book. Research programs on Hepatitis C, BSE, SARS, Avian flu and current vaccination policies all developed along the same logic, that of maximizing financial profits. Whenever we try to understand how some highly questionable therapeutic policies 13 Foreword I have been recommended at the highest levels of public health authorities (WHO, CDC, RKI etc.), we frequently discover either embarrassing conflicts of interests, or the lack of essential control experiments, and always the strict rejection of any open debate with authoritative scientists presenting dissident views of the pathological processes. Manipulations of statistics, falsifications of clinical trials, dodging of drug toxicity tests have all been repeatedly documented. All have been swiftly covered up, and none have been able to, so far, disturb the cynical logic of today's virus research business. The cover-up of the neurotoxicity of the mercury containing preservative thimerosal as a highly probable cause of autism among vaccinated children apparently reached the highest levels of the US govemement ... (see article "Deadly Immunity" from Robert F. Kennedy Jr. in chapter 8) Virus Mania is a social disease of our highly developed society. To cure it will require conquering fear, fear being the most deadly contagious virus, most efficiently transmitted by the media. Errare humanum est sed diabolicum preservare . - .. (to err is human, but to preserve an error is diabolic). Etienne de Harven, MD Professor Emeritus of Pathology at the University of Toronto and Member of the Sloan Kettering Institute for Cancer Research, New York (1956 - 1981) Member of Thabo Mbeki's AIDS Advisory Panel of South Africa
  5. excellent discussion going on here, from my understanding a body cannot get sick normally if its alkaline, most dis=ease is cause as the diet is to acidic which in a round about way has already been pointed out here. I learnt this from my teacher lol, if your a heavy meat eater for example your body is acidic. add stress lack of movement stale air bad diets what you have is a prime overload of acid and guess what these fuckers did for last few years., yeah we already know enjoying this discussion kind regards
  6. as a mischievous teen in the 80s we built our own FM radios to do pirate radio pantec kits if I remember correctly and yes they worked very well only needed a radio receiver to listen which most houses have very easily done, if you wish for community conversations a cobra 148 cb radio swap out the crystal's to improve the distances
  7. that is pure class bro i friggin spat my coffee
  8. I was laughing at the woman behind him proper couldn't contain herself saw that before great though.
  9. i am on it as my sisters fella unjabbed against it. admitted to hospital with a ear infection in IRELAND within days was on a ventilator now deceased. Ive asked my sister to obtain his medical records from the hospital, need to know what treatment he was getting to deteriorate so frigging quickly he didn't test positive for covid on entry to hospital. its a waiting game i assume, not the quickest to get info out of any hospital he was in his 50s physically active.
  10. Liverpool echo.co.uk/news/uk-world-news/what-boris-Johnsons-living-covid-22700294 The Government is said to be drawing up plans for how we "live with covid" long-term without the need for any new restrictions. Boris Johnson is expected to reveal the plan for how the UK can live with the virus in the future, setting out what restrictions we will see in the months ahead. The Prime Minister said: "It is pointless keeping giving more and more vaccines to people who are not going to get very ill. We should just let them get ill and deal with that." full article on link above, so it maybe its back tracking time now or not cos they change like the wind.
  11. actual wording bro Small increases in Roche N seropositivity have recently been observed across all age groups (Table 6) compared to the previous 12-week period. Increases in the overall COVID-19 case rates in England have been observed across all age groups and regions in week 40 (Weekly national Influenza and COVID-19 surveillance report week 41). Roche S seropositivity in blood donors has plateaued and is now over 96% across all age groups. Seropositivity estimates for S antibody in blood donors are likely to be higher than would be expected in the general population and this probably reflects the fact that donors are more likely to be vaccinated. Seropositivity estimates for N antibody will underestimate the proportion of the population previously infected due to (i) blood donors are potentially less likely to be exposed to natural infection than age matched individuals in the general population (ii) waning of the N antibody response over time and (iii) recent observations from UK Health Security Agency (UKHSA) surveillance data that N antibody levels appear to be lower in individuals who acquire infection following 2 doses of vaccination. Vaccination has made an important contribution to the overall Roche S increases observed since the roll out of the vaccination programme, initially amongst individuals aged 50 years and above who were prioritised for vaccination as part of the phase 1 programme and more recently in younger adults as part of phase 2 of the vaccination programme. Roche S levels by age group and month The Roche S assay that the UK Health Security Agency (UKHSA) uses for serological surveillance is fully quantitative, meaning that it measures the level of antibodies in a blood sample; an antibody level above 0.8 AU/ml (approximately one IU/ml using the WHO standard) is deemed positive. The PHE and UKHSA surveillance over the past few months has found that over 97% of the population of blood donors test positive for S-antibodies, which may have resulted from either COVID-19 infection or vaccination. With such high seropositivity, it is important to look at population antibody levels in order to assess the impact of the vaccination booster programme. Figure 6 shows monthly categorised Roche S levels in N-antibody negative individuals by age group. Almost all tested S-antibody negative during December. In the 3 oldest age groups, the impact of first vaccine dose, then second vaccine dose, can be seen from December through June, as the profile of population antibody levels increases. Then from June through September the profile of antibody levels in these cohorts gradually decreases, consistent with waning. During October there is a small increase in percentage of donors with high antibody levels of 1000+ AU/ml for the 70 to 84 age group only, following the initiation of the booster programme. The higher profile of antibody levels in the youngest age group, is likely a result of a combination of factors including stronger immune responses in younger individuals and the higher antibody levels produced after mRNA vaccination. Figure 7 shows categorised Roche S levels in N-antibody positive individuals, those likely to have experienced past infection. Pre-vaccination antibody levels will be influenced by time since infection, variant and severity of infection, as well as personal factors such as underlying health conditions and age. At the start of the vaccination rollout in December antibody levels typically sat within the range of 0.8 to 1000 AU/ml, after vaccination antibody levels typically exceed 1000 AU/ml. Comparing Figure 6 with Figure 7, the overall higher profile of antibody levels in those who have experienced past infection is evident; both vaccination post infection and breakthrough infection following vaccination are expected to boost existing antibody levels. Researchers across the globe are working to better understand what antibody levels mean in terms of protection against COVID-19. Current thinking is that there is no threshold antibody level that offers complete protection against infection, but instead that higher antibody levels are likely to be associated with lower probability of infection.https://assets.publishing.service.gov.uk/government/uploads/system/uploads/attachment_data/file/1027511/Vaccine-surveillance-report-week-42.pdf
  12. you are right on the infinity part, now the hexagon a cell? part of piece, or another way of putting it the human cell is in need of constant meds for infinity the vaxed anyway that's my thoughts. as the hexagon can if enough pieces can make the whole. or if hollow can make a net. cyber net maybe? sorry thinking out loud.
  13. nar mate you tough cookie, takes a man to be honest. i tip my hat to you
  14. i would crack a few stink bombs see how quick store empty's they would quickly realise masks don't work
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